Turn It In

Image from Turnitin.com

As you write your Lecture Reviews, Book Reviews, and Journal Article Reviews you will submit your writing to Turn It In to check for potential plagiarism.

To help you avoid the problem of plagiarism, we will be using “Turnitin” through Canvas. This is an online tool that you can use to detect possible problems with plagiarism and correct them before you submit your work.

For more information on the topic of plagiarism, check out the following resources:
>> Turnitin Student Quickstart Guide
>> Plagiarism.org
>> Plagiarism at Wikipedia

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2 Responses to Turn It In

  1. Ethan Bessette says:

    Lecture Review attached…

  2. Ethan Bessette says:

    Ethan Bessette
    Hour 1
    Prof. Robert A. Weinberg
    Lecture 24: AIDS
    Fall 2004

    The topic the lecture covers is AIDS. AIDS is a disease that takes place in the immune system, and interferes with the body’s ability to fight infections. T helper cells are apart of the process. Scientist observes T helper cells to be on their surface, this blows them up to a much larger size. T helper cells are generated through antibody like genes being moved around the body system. TH is the name of the function of T helper cells. Only the functions sense the presence of antigens. TH senses antigens by using the MHC class 2. The MHC class 2 has two cells involved, a macrophage cell, or a dendritic cell. The MHC class 2 molecules getting exposed to the T helper cells plays a big part to how AIDS are developed. The reason why the T helper cell connects to the MHC class 2 is due to their T cell receptor. After a while, T helper cells go through a “clonal expansion,” and are now activated. The activations of the AIDS disease are the reaction T helper cells have on the antigen-presenting cell. A third substance that is involved is a B cell. The B cell, like the T cell, is on its surface. Scientist have no clue how this B cell functions which is one of many reasons why scientist are yet to find a cure. After the T cell helpers encounter with the MHC class 2 cells, The T helper cell starts to grow and multiplies in the area. Then, the B cells have a chance to react when the T helper cell recognizes the B cell’s antigen. After that has occurred, then the B cell eventually starts to make multiple IGM molecules. IGM is the name of the antibody. This is a very complicated process. The reason why it is so complicated is because the immune system makes it complicated on purpose. It is extremely important the immune system doesn’t make antibodies. Antibodies are bad for the immune system because they are self reacted. Self-reacted particles are bad in the immune system because it creates all sorts of diseases in the immune system. After that process, it stays permanent with its carrier. That is the disease of AIDS. The AIDS virus was first called human immunodeficiency virus. AIDS were first discovered in 1981 in San Francisco. The disease was sexual transmitted through five men. The reason why the scientist named the disease AIDS was because the men were diagnosed with all sorts of genital diseases. Around the time it was found was also the time it started to rapidly spread. Scientist called AIDS an “infectious agent.” The scientific community spent a lot of years trying to figure out what was causing AIDS. When the body goes through the functions with the T helper cells regular functions plus the sexual transmitted diseases, it devolves another disease called HIV. HIV cannot be cured; the HIV attacks and replicated the T helper cells which modern technology cannot do anything to stop it. When a person is HIV positive, they get treatment. The treatment is not efficient, when people with HIV take the treatment; it just gives them time before the real deal hits, which is AIDS. The Immune system attempts to take out the HIV. It does this by killing all the T helper cells that are involved in the production of HIV. This takes a lot of work for the immune system, which makes it vulnerable to other diseases. That is the overall function on how AIDS works, it weakens our immune system to the point where it cannot fight off other diseases. Once an individual is infected, that individual is infected until the day they die. So far there is no way of getting rid of AIDS because it is a viral infection. A viral infection is an infection cause by a virus and can’t be treated with any antibiotics. Other viral infections include herpes, chicken pox, and the common cold. There is no cure for AIDS, but there is a certain drug that slows down the process. These drugs are called Antiretroviral drugs (ARV’s). These drugs may put the infection on hold, but the virus is still hiding in the body.

    I thought this video was very informational. The instructor seemed to really know what he was talking about. I know that AIDS is a sexually transmitted disease, which makes me think it can be simply avoided. From the knowledge I learned about AIDS so far in my life, most people don’t know they have it until they are tested. Protection helps people not to get the disease, but they don’t always work. So the reason why I think AIDS can be avoided other than protection and knowing, is to know what you are getting into in the first place. That is what I thought about AIDS. After watching a video lecture from Professor Robert A. Weinberg about AIDS, I really got to know how it works. The function of T helper cells, MHC class 2, and B cells gives me an insight on how the body functions without the disease versus how it functions while carrying the disease. This particular video is hard to relate to what we learned in class so far, but I think I can connect it to one topic. We have talked about cells, what they do for the body’s function, this connects because what we talked about how certain cells carry over certain information to other cells. This connects to how the T helper cells carrying the disease through certain body functions. We also ran experiments on how cells react to certain conditions, which can also be connected to how the cells react while carrying the HIV. My overall take to AIDS is not about how it works, but how having that disease can change a human’s life so much, and so far at my age, I choose not to think about it outside of education.

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